简介：Usingtransganglionicdegenerativemethed,weinvestigatedtheprimaryafferentinformationofacupointsofQuanliao(SI18)andShibai(ST2).Theresultsshowedthatthelargeamountsofdegenerativefiberscouldbefoundbilaterallyintrigeminalspinalnucleusanditstract,trigeminalprincipalsensorynucleus,trigeminalmesencephalicnucleusandsomedegenerationscouldbeobservedipsilaterallyinsolitarytractanditsnucleus,medullarylateralreticularnucleus,giganto-cellularreticularnucleusandnucleusraphemagnusfollowingtransectionofinfraorbitalnerve.Averymoderatedegenerativefiberscouldbeidentifedinipsilateralaccessoryspinalnucleusandspinalreticu-larnucleus.Afewdegenerativefiberswereseenonthecontralateralmagnocellularpartofcaudatepartoftrigeminalspinalnucleusbypassingthroughtheposteriorcommissureofcanaliscentralis.TheseresultssuggestthattheprimaryinformationofacupointsofQuanliaoandShibaicouldbetrans-mitteredintotheabovestruc

简介：Inthispresentstudy,theruodeloftherabbitwithacutemyocardialischemia（AMI）wasusedtoexploremechanismsofinterrelationhetweenNeiguanacupoint（PC6）ofthePeri-cardiumMeridianandthehearthyemployingphysiologicalandmorphologicaImethods.Itwasdemonstratedthatelectroacupuncture（EA）atNeiguan（PC6）couIdraisetheeIectricalexcitahilityofischemicmyocardium,lessenthedispersityofrecoveryexcitability,correctthedisorderofelectricalactivity;protectmyocardiacglycogenandphosphorylaseandalleviatetheirdepletion,increasecontentofribonucleiacid（RNA）,etc.,indicatingthatEAcouldhelpoxidativernetaholismcarryoutnormal-IyandirnProvenutritionalstateoftheischemicmyocardiurn.ThekeypointofEAactionisprobablyregulatesthecoronarymicroclrculationofheartandreclistributesmyocardialbloodflow.Studieswithalkalinephosphatase（ALP）,Mg2--ATPaseandABScastingoftheIeftcoronaryarteryshowedthatafterEAthenumberandtheIengthof